Nos principales publications
- Chatzifrangkeskou, M., Yadin, D., Marais, T., Chardonnet, S., Cohen-Tannoudji, M., Mougenot, N., Schmitt, A., Crasto, S., Di Pasquale, E., Macquart, C., et al. (2018). Cofilin-1 phosphorylation catalyzed by ERK1/2 alters cardiac actin dynamics in dilated cardiomyopathy caused by lamin A/C gene mutation. Hum. Mol. Genet. 27, 3060–3078.
- Giordani, L., He, G.J., Negroni, E., Sakai, H., Law, J.Y., Siu, M.M., Wan, R., Tajbakhsh, S., Cheung, T.H., and Le Grand, F. (2019). High-dimensional single-cell cartography reveals novel skeletal muscle resident cell populations. Molecular Cell in press.
- Lacour, F., Vezin, E., Bentzinger, C.F., Sincennes, M.-C., Giordani, L., Ferry, A., Mitchell, R., Patel, K., Rudnicki, M.A., Chaboissier, M.-C., et al. (2017). R-spondin1 Controls Muscle Cell Fusion through Dual Regulation of Antagonistic Wnt Signaling Pathways. Cell Rep 18, 2320–2330.
- Le Dour, C., Macquart, C., Sera, F., Homma, S., Bonne, G., Morrow, J.P., Worman, H.J., and Muchir, A. (2017). Decreased WNT/β-catenin signalling contributes to the pathogenesis of dilated cardiomyopathy caused by mutations in the lamin a/C gene. Hum. Mol. Genet. 26, 333–343.
- Parisi, A., Lacour, F., Giordani, L., Colnot, S., Maire, P., and Le Grand, F. (2015). APC is required for muscle stem cell proliferation and skeletal muscle tissue repair. J. Cell Biol. 210, 717–726.
- Rodriguez, B.M., Khouzami, L., Decostre, V., Varnous, S., Pekovic-Vaughan, V., Hutchison, C.J., Pecker, F., Bonne, G., and Muchir, A. (2018). N-acetyl cysteine alleviates oxidative stress and protects mice from dilated cardiomyopathy caused by mutations in nuclear A-type lamins gene. Hum. Mol. Genet. 27, 3353–3360.
- Rudolf, A., Schirwis, E., Giordani, L., Parisi, A., Lepper, C., Taketo, M.M., and Le Grand, F. (2016). β-Catenin Activation in Muscle Progenitor Cells Regulates Tissue Repair. Cell Rep 15, 1277–1290.
- Vignier, N., Chatzifrangkeskou, M., Morales Rodriguez, B., Mericskay, M., Mougenot, N., Wahbi, K., Bonne, G., and Muchir, A. (2018). Rescue of biosynthesis of nicotinamide adenine dinucleotide protects the heart in cardiomyopathy caused by lamin A/C gene mutation. Hum. Mol. Genet. 27, 3870–3880.
